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Transgenic Mice
Transgenic Mice
Striatal and nigral pathology in a lentiviral rat model of Machado-Joseph disease
Genetics / Animal Behavior / Gene expression / Transgenic Mice / Biological Sciences / Visual Cortex / Cell line / Humans / Mice / Female / Animals / Male / Human Molecular Genetics / Lentivirus / Substantia nigra / Rat Brain / Aged / Middle Aged / Rats / Wistar Rats / Rat Model / Machado-Joseph Disease / Visual Cortex / Cell line / Humans / Mice / Female / Animals / Male / Human Molecular Genetics / Lentivirus / Substantia nigra / Rat Brain / Aged / Middle Aged / Rats / Wistar Rats / Rat Model / Machado-Joseph Disease
BIOLOGIA MOLECULAR Y MEDICINA A FINES DEL SIGLO XX
Molecular Biology / Medicine / Transgenic Mice
Vascular Endothelial Cell-Specific NF-B Suppression Attenuates Hypertension-Induced Renal Damage
Atherosclerosis / Kidney diseases / Inflammation / Transgenic Mice / Signal Transduction / Humans / Hypertension / Endothelial Cells / High Blood Pressure / Mice / Blood Pressure / Nitric oxide / Animals / NF-kappa B / Clinical Sciences / Endothelial cell / Tumor necrosis factor-alpha / Circulation / Albuminuria / Angiotensin II / Methyl Ester / nuclear factor kappa B / Gene Expression Regulation / Humans / Hypertension / Endothelial Cells / High Blood Pressure / Mice / Blood Pressure / Nitric oxide / Animals / NF-kappa B / Clinical Sciences / Endothelial cell / Tumor necrosis factor-alpha / Circulation / Albuminuria / Angiotensin II / Methyl Ester / nuclear factor kappa B / Gene Expression Regulation
In vivo staphylococcal superantigen-driven polyclonal Ig responses in mice: dependence upon CD4+ cells and human MHC class II
Immunology / Autoimmunity / Transgenic Mice / Spleen / Humans / Mice / Enterotoxins / Animals / Staphylococcus aureus / MHC class II / Model System / Species Specificity / Knockout Mice / immunoglobulin G / STAPHYLOCOCCAL-ENTEROTOXIN-B / Mice / Enterotoxins / Animals / Staphylococcus aureus / MHC class II / Model System / Species Specificity / Knockout Mice / immunoglobulin G / STAPHYLOCOCCAL-ENTEROTOXIN-B
Acute Leukemia with Promyelocytic Features in PML/RARalpha Transgenic Mice
Aging / Transcription Factors / Multidisciplinary / Hematopoiesis / Transgenic Mice / Spleen / Hematopoietic Stem Cells / Acute Myeloid Leukemia / Cell Differentiation / Humans / Mice / Animals / Polymerase Chain Reaction / Bone marrow / First Year / Retinoic Acid / Tretinoin / Lymphocytes / Reference Values / Fusion Protein / Acute Promyelocytic Leukemia / Retinoic Acid Receptor / Spleen / Hematopoietic Stem Cells / Acute Myeloid Leukemia / Cell Differentiation / Humans / Mice / Animals / Polymerase Chain Reaction / Bone marrow / First Year / Retinoic Acid / Tretinoin / Lymphocytes / Reference Values / Fusion Protein / Acute Promyelocytic Leukemia / Retinoic Acid Receptor
Loss of Prion Protein in a Transgenic Model of Amyotrophic Lateral Sclerosis
Cognitive Science / Protein Folding / Skeletal muscle biology / Motor neuron / Transgenic Mice / Amyotrophic Lateral Sclerosis / Molecular chaperones / Molecular and cellular biology / Genetic Testing / Mutation / Mice / Animals / Spinal Cord / Superoxide Dismutase / Western blot / Sciatic Nerve / Cellular and Molecular Neuroscience / Enzymatic Activity / Neurosciences / Down-Regulation / Prion Protein / DNA mutational analysis / Amyotrophic Lateral Sclerosis / Molecular chaperones / Molecular and cellular biology / Genetic Testing / Mutation / Mice / Animals / Spinal Cord / Superoxide Dismutase / Western blot / Sciatic Nerve / Cellular and Molecular Neuroscience / Enzymatic Activity / Neurosciences / Down-Regulation / Prion Protein / DNA mutational analysis
Transcription factor IRF4 controls plasma cell differentiation and class-switch recombination
Immunology / Flow Cytometry / Transcription Factors / Transgenic Mice / Cell Differentiation / Mice / Female / Animals / Male / Class switch recombination / Transcription Factor / B Lymphocytes / DNA binding proteins / Antigens / immunoglobulin G / Germinal Center / Gene Expression Regulation / Activation Induced Deaminase / Mice / Female / Animals / Male / Class switch recombination / Transcription Factor / B Lymphocytes / DNA binding proteins / Antigens / immunoglobulin G / Germinal Center / Gene Expression Regulation / Activation Induced Deaminase
Developmental consequences of in utero sodium arsenate exposure in mice with folate transport deficiencies
Reproduction / Arsenic / Folic acid / Transgenic Mice / Mice / Female / Animals / Male / Genetic modification / Detoxification / Membrane transport proteins / Genetically Modified / Genotype / Chimera / Reproductive Toxicity / Arsenates / Female / Animals / Male / Genetic modification / Detoxification / Membrane transport proteins / Genetically Modified / Genotype / Chimera / Reproductive Toxicity / Arsenates
Micro-dystrophin cDNA ameliorates dystrophic phenotypes when introduced into mdx mice as a transgene
Skeletal muscle biology / Fluorescence Microscopy / Gene Therapy / Immunohistochemistry / Western blotting / Transgenic Mice / Humans / Mice / Animals / Adeno-associated virus / Biochemical / Duchenne Muscular Dystrophy / Creatine Kinase / Phenotype / Skeletal Muscle / Time Factors / Dystrophin / Biochemistry and cell biology / Gene Transfer / Transgenic Mice / Humans / Mice / Animals / Adeno-associated virus / Biochemical / Duchenne Muscular Dystrophy / Creatine Kinase / Phenotype / Skeletal Muscle / Time Factors / Dystrophin / Biochemistry and cell biology / Gene Transfer
La proteína relacionada con la paratirina: un nuevo mediador en la nefropatía diabética
Molecular Mechanics / Transgenic Mice / Logistic Regression Analysis / Diabetic Nephropathy / Cell Cycle regulation / In Vitro Studies / Angiotensin II / Renal disease / Experimental Model / In Vitro Studies / Angiotensin II / Renal disease / Experimental Model
La proteína relacionada con la paratirina: un nuevo mediador en la nefropatía diabética
Molecular Mechanics / Transgenic Mice / Logistic Regression Analysis / Diabetic Nephropathy / Cell Cycle regulation / In Vitro Studies / Angiotensin II / Renal disease / Experimental Model / In Vitro Studies / Angiotensin II / Renal disease / Experimental Model
Transgenic expression of human acetylcholinesterase induces progressive cognitive deterioration in mice
Aging / Molecular Mechanics / Transgenic Mice / Biological Sciences / Learning and Memory / Humans / Mice / Animals / Cell Death / Acetylcholinesterase / Open Field / Central Nervous System / Animal Model / Acetylcholine / Spatial Learning / Neurons / Cholinesterase inhibitors / Structural Change / Enzyme / Locomotor Activity / Neuromuscular Junction / Transgene Expression / Cognition disorders / Water maze / Humans / Mice / Animals / Cell Death / Acetylcholinesterase / Open Field / Central Nervous System / Animal Model / Acetylcholine / Spatial Learning / Neurons / Cholinesterase inhibitors / Structural Change / Enzyme / Locomotor Activity / Neuromuscular Junction / Transgene Expression / Cognition disorders / Water maze
Prostate targeting: PSP94 gene promoter/enhancer region directed prostate tissue-specific expression in a transgenic mouse prostate cancer model
Clinical Trial / Gene Therapy / Immunohistochemistry / Prostate Cancer / Cell Culture / Transgenic Mice / Biological Sciences / Mice / Animals / Male / Gene Targeting / Disease Progression / Amino Acid Profile / Transgenic Mouse Technology / Tumor Progression / Tissue Specificity / Transgenic Mice / Biological Sciences / Mice / Animals / Male / Gene Targeting / Disease Progression / Amino Acid Profile / Transgenic Mouse Technology / Tumor Progression / Tissue Specificity
DNA repair efficiency in transgenic mice over expressing ribosomal protein S3
Oxidative Stress / DNA damage / DNA repair / Transgenic Mice / Protein synthesis / Humans / Mutation / Mice / Female / Animals / Male / Ribosomal proteins / Oxidative Damage / In Vitro Studies / Oxidative Dna Damage / Ribosomal Protein / Binding affinity / Humans / Mutation / Mice / Female / Animals / Male / Ribosomal proteins / Oxidative Damage / In Vitro Studies / Oxidative Dna Damage / Ribosomal Protein / Binding affinity
Vascular Endothelial Cell Specific NF- B Suppression Attenuates Hypertension-Induced Renal Damage
Atherosclerosis / Kidney diseases / Inflammation / Transgenic Mice / Signal Transduction / Humans / Hypertension / Endothelial Cells / High Blood Pressure / Mice / Blood Pressure / Nitric oxide / Animals / NF-kappa B / Clinical Sciences / Endothelial cell / Tumor necrosis factor-alpha / Circulation / Albuminuria / Angiotensin II / Methyl Ester / nuclear factor kappa B / Gene Expression Regulation / Humans / Hypertension / Endothelial Cells / High Blood Pressure / Mice / Blood Pressure / Nitric oxide / Animals / NF-kappa B / Clinical Sciences / Endothelial cell / Tumor necrosis factor-alpha / Circulation / Albuminuria / Angiotensin II / Methyl Ester / nuclear factor kappa B / Gene Expression Regulation
Vascular Endothelial Cell-Specific NF-B Suppression Attenuates Hypertension-Induced Renal Damage
Atherosclerosis / Kidney diseases / Inflammation / Transgenic Mice / Signal Transduction / Humans / Hypertension / Endothelial Cells / High Blood Pressure / Mice / Blood Pressure / Nitric oxide / Animals / NF-kappa B / Clinical Sciences / Endothelial cell / Tumor necrosis factor-alpha / Circulation / Albuminuria / Angiotensin II / Methyl Ester / nuclear factor kappa B / Gene Expression Regulation / Humans / Hypertension / Endothelial Cells / High Blood Pressure / Mice / Blood Pressure / Nitric oxide / Animals / NF-kappa B / Clinical Sciences / Endothelial cell / Tumor necrosis factor-alpha / Circulation / Albuminuria / Angiotensin II / Methyl Ester / nuclear factor kappa B / Gene Expression Regulation
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